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Istituto Veneto di Scienze, Lettere ed Arti - Testata per la stampa

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International Workshop on Viruses, Genes and Cancer

International Workshop on Viruses, Genes and Cancer

22-23 March 2007
Istituto Veneto di Scienze, Lettere ed Arti
Palazzo Cavalli Franchetti

Organised by
Istituto Veneto di Scienze, Lettere ed Arti
Department of Oncology and Surgical Sciences, University of Padova
Veneto Institute of Oncology (IOV),
IRCCS, Padova

Main Topics
* Viruses as etiologic agents of cancer: a 2007 appraisal
* Host susceptibility and resistance to viruses and cancer
* Immunosurveillance and immunoregulation in virus infection and in virus-associated cancer
* The aids epidemic
* aids vaccines and therapeutics
* Genetics and epigenetics of hematologic
* Pathogenesis of htlv-associated diseases
* The future of cancer research and therapy

Scientific Committee

Padova University and IVSLA
Ohio State University, Columbus and Ferrara University
Padova University and IVSLA
Columbia University, New York and Padova University
National Cancer Institute, NIH Bethesda-Maryland
Dana Farber Institute, Harvard University, Boston

Organizing Committee
Department of Oncology and Surgical Science, University of Padova and Veneto
Institute of Oncology, IRCCS, Padova
via Gattamelata 64, 35128 Padova
ph +39 049 821 5804
mail albido@unipd.it

It is estimated that approximately 17% of human cancers are linked to infectious agents. Beginning with the identification of Epstein- Barr virus in Burkitt lymphoma cells in 1964, intensive research into the connections between viral infections and cancer have led to the identification of 6 human tumor viruses, and continued research is likely to uncover new agents. Dissection of the interactions between human tumor viruses and host cells have yielded significant advances in our understanding of how these oncogenic infectious agents elude the immune system, persist in their hosts, and promote the stepwise chain of events that result in neoplastic transformation. Human tumor virology has also provided fundamental knowledge of pathway governing cell proliferation, led to the identification of oncogenes, and contribued to the understanding of tumor suppressor function. Information gained from these studies has been applied to develop vaccines against hepatitis B virus and human papillomavirus, which will in the long term greatly reduce cases of hepatocellular and cervical carcinoma. Continued study of the human tumor viruses will be essential for the development of additional preventative vaccines and for designing strategies to eliminate established infections and counteract the particular risk that human tumor viruses pose in the context of immunosuppression, in particular AIDS.


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